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English Wikipedia references for Jleukbio.org 1-20 of 28
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Autism
Autism is a brain development disorder characterized by impaired social interaction and communication, and by restricted and repetitive behavior. These signs all begin before a child is three years old.
Autism
Signal transduction
biology, 'transduction'cell converts one kind of signal or stimulus into another. Most processes of signal transduction involve ordered sequences of biochemical reactions inside the cell, which are carried out by enzymes, activated by second messengers, resulting in a signal transduction pathway.
Signal_transduction
Phagocyte
Phagocytes are the white blood cells that protect the body by ingesting (phagocytosing) harmful foreign particles, bacteria and dead or dying cells. They are essential for fighting infections, and for subsequent immunity. Phagocytes are important throughout the animal kingdom, and are highly developed in vertebrates.
Phagocyte
Endothelium-derived relaxing factor
Talk:Endothelium-derived_relaxing_factor
Proteinase 3
Proteinase 3 also known as PRTN3 is an enzyme which in humans is encoded by the PRTN3 gene.
Proteinase_3
CCL2
For the ICAO airport code see Candle Lake Airpark, for the diradical compound see Dichlorocarbene.Chemokine (C-C motif) ligand 2 (CCL2) is a small cytokine belonging to the CC chemokine family that is also known as monocyte chemotactic protein-1 (MCP-1). CCL2 recruits monocytes, memory T cells, and dendritic cells to sites of tissue injury and infection.
CCL2
Causes of autism
Autism and autism spectrum disorders are complex neurodevelopmental disorders. Many causes of autism have been proposed, but its theory of causation is still incomplete.
Causes_of_autism
TGN1412
TGN1412 (also known as CD28-SuperMAB) is the working name of an immunomodulatory drug which was withdrawn from development, originally intended for the treatment of B cell chronic lymphocytic leukemia (B-CLL) and rheumatoid arthritis. It is a humanised monoclonal antibody that not only binds to, but is a strong agonist for, the CD28 receptor of the immune system's T cells.In its first human clinical trials, in March 2006, it caused catastrophic systemic organ failure in the subjects, despite being administered at a supposed sub-clinical dose of 0.113 March 2006.
TGN1412
RANK
RANK (Receptor Activator of Nuclear Factor κ B), also known as TRANCE Receptor, is a type I membrane protein that is expressed on the surface of osteoclasts and is involved in the activation of osteoclasts upon ligand binding. RANK is also expressed on dendritic cells and facilitates immune signaling. RANK ligand is found on the surface of stromal cells, osteoblasts, and T cells.
RANK
Interleukin-1 receptor
Interleukin-1 receptor (IL-1R) is a cytokine receptor which binds interleukin 1. Two forms of the receptor exist. The type I receptor is primarily responsible for transmitting the inflammatory effects of interleukin-1 (IL-1) while type II receptors may act as a suppressor of IL-1 activity by competing for IL-1 binding.
Interleukin-1_receptor
Bactericidal/permeability increasing protein
Bactericidal/permeability increasing protein (BPI) is a 456 residue (~50kDa) protein which is part of the innate immune system.
Bactericidal/permeability_increasing_protein
Interleukin 16
Interleukin 16 (lymphocyte chemoattractant factor), also known as IL16, is a human gene. This gene was discovered in 1982 at Boston University by Dr. David Center and Dr. William Cruikshank.Interleukin 16 (IL-16) is a cytokine that released by a variety of cells (including lymphocytes and some epithelial cells) that has been characterized as a chemoattractant for certain immune cells expressing the cell surface molecule CD4.
Interleukin_16
Interleukin 21
Interleukin 21, also known as IL21, is a human gene.The protein encoded by this gene, Interleukin 21, a cytokine that has potent regulatory effects on cells of the immune system, including natural killer (NK) cells and cytotoxic T cells that can destroy virally infected or cancerous cells.
Interleukin_21
Interleukin 17
Interleukin-17 (IL-17, or IL-17A) is the founding member of a group of cytokines called the IL-17 family. IL-17A, was originally identified as a transcript from a rodent T-cell hybridoma by Rouvier et al. in 1993. Known as CTLA8 in rodents, IL-17 shows high homology to viral IL-17 encoded by an open reading frame of the T lymphotropic rhadinovirus Herpesvirus saimiri.
Interleukin_17
Autism Research Institute
The Autism Research Institute (ARI), established in 1967 by Bernard Rimland, is a San Diego, California, based nonprofit that funds research and provides information on autism and autism spectrum disorders. Stephen M. Edelson became the director of ARI upon Rimland's death in 2006.The ARI holds that autism can be treated through a combination of intensive behavior modification, such as Applied Behavior Analysis, and a wide variety of biomedical interventions, including the use of drugs, dietary supplements, special diets, and chelation therapy.
Autism_Research_Institute
CXCL1
Chemokine (C-X-C motif) ligand 1 (CXCL1) is a small cytokine belonging to the CXC chemokine family that was previously called GRO1 oncogene, GROα, KC, Neutrophil-activating protein 3 (NAP-3) and melanoma growth stimulating activity, alpha (MSGA-α). In humans, this protein is encoded by the CXCL1 gene.
CXCL1
Polyclonal B cell response
[[
Polyclonal_B_cell_response
NONOate
In chemistry, a NONOate is a compound having the chemical formula R1R3N-(NO−)-N=O, where R1 and R2 are alkyl groups. One example for this is 1,1-diethyl-2-hydroxy-2-nitrosohydrazine, or diethylamine dinitric oxide. These compounds are unusual in having three sequential nitrogen atomsamine functional group, a bridging NO− group, and a terminal nitrosyl group. In contact with water, these compounds release NO (nitric oxide).
NONOate
CD33
CD33 is a transmembrane receptor expressed on cells of myeloid lineage. It is usually considered myeloid-specific, but it can also be found on some lymphoid cells.It binds sialic acids, therefore is a member of the SIGLEC family of lectins.
CD33
Reference desk/Archives/Science/2007 May 10
Wikipedia:Reference_desk/Archives/Science/2007_May_10